Which of the following is NOT a classic precipitant of hemolytic crisis in G6PD deficiency?

In the context of G6PD deficiency, a hemolytic crisis occurs when red blood cells are subjected to oxidative stress due to various triggers. Glucose-6-phosphate dehydrogenase (G6PD) plays a crucial role in protecting red blood cells from oxidative damage. The classic precipitants of hemolytic crises include infections, certain medications, and dietary elements like fava beans.

Infections can lead to hemolysis due to the increased metabolic demands on red blood cells and the generation of reactive oxygen species during the immune response. Fava beans contain oxidative compounds that can directly damage red blood cells in individuals with G6PD deficiency, leading to hemolysis. Sulfonamide antibiotics are known to cause oxidative stress and trigger hemolytic anemia in susceptible patients.

Beta blockers, on the other hand, are not recognized as classic precipitants of hemolytic crises in G6PD deficiency. While certain medications can induce hemolysis, beta blockers are not typically associated with this phenomenon. Therefore, when considering the known triggers of hemolytic crises in G6PD deficiency, beta blockers stand out as the option that does not fit the classic pattern.